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Furthermore, the authors pointed out a possible connection between Nr4a1 and Gpx1 transcription, indicating that the antioxidant properties of NAC may be partially mediated through the modulation of stress-responsive transcription factors

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Dual inhibition of the MEK-ERK pathway (classical tumor signaling pathway) and Gln metabolism can induce redox stress, as evidenced by reduced mitochondrial membrane potential, elevated ROS levels, and inhibition of Gln degradation, and may be an effective therapeutic strategy for KRAS -driven NSCLC (82)

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